Buy Nolvadex Post Cycle Therapy

Buy Nolvadex Post Cycle Therapy

Post Cycle Therapy

A post cycle therapy plan or “PCT”, it’s a phrase that’s often thrown around inappropriately on many steroid message boards. In many cases, people expect way too much out of post cycle therapy, and others won’t give it a chance based on a lack of understanding. With this in mind, we want to explain the purpose of post cycle therapy, what you can actually expect and the best way to implement it. Further, we want to discuss when it should be implemented; in some cases, a PCT plan will be followed when it shouldn’t have been; don’t worry, this will all make sense.

What is a PCT Plan?

When we supplement with anabolic androgenic steroids. our natural hormone levels are altered. Most anabolic steroids suppress our natural testosterone production to one degree or another, and if we’re not careful our estrogen and progesterone levels can increase beyond a healthy range. Of course, estrogen and progesterone can both be controlled while on cycle with proper supplementation practices, but the testosterone suppression will remain. Then we reach the point where our cycle has come to an end; we have discontinued the use of all anabolic steroids. and as a result something must be done. When we discontinue our steroid use. our testosterone levels are still in a suppressed state, and it’s often recommended you stimulate natural production and let your body normalize. While testosterone stimulation is the primary purpose, the normalization factor of a post cycle therapy plan is greatly important. Of course, as eluded to early on, sometimes implementing a PCT isn’t the best idea, and will delve into that shortly.

What to Expect

The primary purpose of post cycle therapy is to stimulate your natural production of testosterone and shorten or enhance the total recovery process. Understand this here and now; there is no post cycle therapy plan on earth that can return your natural testosterone levels back to where they were prior to anabolic steroid use. Further, if you supplemented with anabolic steroids improperly and caused severe damage to your HPTA there’s no PCT plan that will help you. In any case, assuming your cycle was of a responsible nature, a post cycle therapy phase will by design stimulate your pituitary to release more Luteinizing Hormone (LH) and Follicle Stimulating Hormone (FSH) which will in-turn stimulate the testicles to produce more testosterone.

Without such a PCT plan, it could easily take a year or more for your natural levels to recover, and this is not only stressful to the body, it can lead to numerous low testosterone symptoms; not to mention it is extremely unhealthy. Conversely, when you implement your post cycle therapy treatment, you will significantly cut down your total recovery time, but there’s something more important. While your natural levels will not be fully recovered, you will have ensured your body has enough testosterone for proper health and function while your levels continue to naturally rise. Sure, you can forgo such a plan if you like, but you’ll only be causing more stress to your body over the long haul, and limiting stress is in part how we define successful performance enhancement; after all, if you’re stressing your body post cycle, this means your steroid use has not be as successful as it could have been.

When to Implement a PCT Plan

It should go without saying; the time to implement post cycle therapy is when all anabolic steroid use has come to an end; this is a given; however, it’s not quite that black and white. If you’re going to be off-cycle for only a short period of time, a PCT plan can be counterproductive and cause even more stress to the body. Remember what we said above; limiting stress is extremely important. If you’re only going to be off-cycle for a short period of time, there’s no reason to stimulate your natural production when you’re going to immediately shut it down again; talk about a shock the body does not appreciate. For this reason, a post cycle therapy period should only be implemented when we’re going to be off-cycle for an extended period of time; meaning, no anabolic androgenic steroids will be present in our system. Of course, the next order of business is to define an extended period of time, and twelve weeks is a good place to start. If you’re going to be off-cycle less than 12 weeks, while you will lose some of your gains they will come back shortly once you go back on-cycle. Conversely, if you’re going to be off-cycle longer than 12 weeks, it’s time for a PCT plan for the reasons discussed above. It must be noted; this time frame of “off-cycle” does not include the PCT period; off means off everything.

Post Cycle Therapy Options

Now that you understand what a post cycle therapy plan is and when and why you should implement it, you need to understand how to implement it and the options you have. How you cycled your anabolic steroids will play a role, but regardless of your steroid use your PCT plan will always include a Selective Estrogen Receptor Modulator (SERM), and Tamoxifen Citrate (Nolvadex ) and Clomiphene Citrate (Clomid ) will always be your best options. Recall what we discussed above in-regards to LH and FSH stimulation; it will be the SERM you use that causes such an action. It really doesn’t matter which SERM you choose, both can get the job done equally as well; simply pick one.

Beyond SERM use, which is essential, we have a few additional options; primarily Human Chorionic Gonadotropin (hCG ). hCG is an extremely powerful peptide hormone that can be used to Clomid prime the body for the SERM therapy to come due to its LH mimicking effect. Of course, hCG abuse can be very dangerous as it is potentially damaging to your HPTA if you use too much or for too long; if you do, your body may become dependent on the mimicked LH. Beyond hcg, another option can be Human Growth Hormone (HGH ) as this will greatly protect your gains made while on-cycle as well as limit body-fat gain that can easily occur post steroid use. While HGH can be useful, you will only be using it if you were using it on-cycle; HGH is something that must be used for extended periods of time, and there’s no point in adding it into a PCT plan that’s only going to last a few weeks.

Now that you understand your options, you need to understand how to implement them. As for HGH, if you used it on-cycle, simply continue with it in the same manner post cycle; nothing changes. Then we have SERM’s which are a must, and the possible addition of hCG. This is where your actual steroid cycle will affect your post cycle therapy plan, and this affect surrounds what types of steroids you used; specifically large and small esters; let’s start with large esters. If your cycle ends with even one large ester anabolic steroid, if you’re only using a SERM you will begin SERM therapy approximately 14-18 days after your last injection. If you’re going to use hCG, you will begin hCG therapy 10 days after your last injection, complete it for 10 days and then begin SERM therapy. As for small esters, if your cycle ends with all small ester based anabolic steroids and you’re only using a SERM you will begin SERM therapy approximately 3 days after your last injection. Conversely, if you’re using hCG, you will begin hCG therapy 3 days after your last injection, complete it for 10 days and then begin SERM therapy.

Now you understand what you need to do and how you need to do it, but you still don’t have the proper doses or full time frame for your post cycle therapy treatment and that’s the final point of our discussion. While Nolvadex and Clomid can work equally as well, they will only work equally as well if they are dosed properly. This is where many fail when they use Clomid as Nolvadex is much stronger on a per milligram basis. For example, with 40mg of Nolvadex, for Clomid to match it you need 150mg. As for hCG dosing, 500iu to 1,000iu per day every day for 10 straight days is your plan and implemented precisely as discussed above. Once the hCG therapy is complete, you will start your Nolvadex therapy at 40mg per day or Clomid at 150mg per day; whichever you choose, you will continue it for two weeks. Once the two weeks is complete, you will complete two more weeks this time with a Nolvadex dosing at 20mg per day or a Clomid dosing at 100mg per day. No, you’re not done yet, you will complete one more week at 10mg per day for Nolvadex or 50mg per day with Clomid and add in an additional week at the same dose if you feel it is necessary.

HCG Pregnyl 5000 IU

Tamoxifen (Med)

PCT - Post Cycle Therapy

Manufacturer: MED Ilac Turkey

Substance: Tamoxifen

After finishing steroid cycle every bodybuilder have the same goal - to keep their body balanced as during the workout. But it is not easy as it feels. After usage of almost all anabolic steroids. production of important hormone, especially testosterone, in your body decrease. And at this time you should start you Post Cycle Therapy. It is considered important key in steroid cycles because it can guaranty an effective result. Without PCT in very short time you can lose considermuscle able tissue that was gained during long workout. With such ancillaries products as Clenbuterol. T3. Proviron. Nolvalex. Tamoximed and others you will quickly restore your natural production of hormone. These substances work as selective estrogen receptor modulator.



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Below are the times you should start your post cycle therapy (PCT for short) depending on the active life of the compound(s) in your steroid stack. Active life refers to the duration of time it takes of the hormone to be absorbed, utilized, and expelled from your system. Although this list is not perfect (as few things can be in something as give and take and dose related as steroid cycling), it is a good guide to include in your research.

Anadrol/Anapolan. 24 hours after last administration

Deca. 21 days after last injection

Dianabo l: 24 hours after last administration

Equipoise. 21 days after last injection

Fina. 3 days after last injection

Primobolan depot. 14 days after last injection

Sustanon. 18 days after last injection

Testosterone Cypionate. 18 days after last injection

Testosterone Enanthate. 14 days after last injection

Nolvadex Post Cycle

Without question Nolvadex post cycle is the primary period of use in-which the individual will benefit from this SERM. For many men it can be the difference in keeping and losing all of their gains. As you understand when you come off an anabolic steroid cycle it can be very hard to maintain the gains you have made. Contrary to general public perception this is not because you’ve built fake muscle; muscle tissue is muscle tissue regardless of what aided in its creation. While there can be many factors as to why we lose some of the gains when we come off a large portion revolves around our hormone levels falling; we no longer possess the total hormone levels we had when we were on cycle. Make no mistake there are other very important factors that come into play but here we are only worried about our hormone levels; specifically testosterone.

When we supplement with anabolic androgenic steroids are natural testosterone is suppressed; for this reason many performance enhancers supplement with the anabolic steroid testosterone while on cycle to ensure their body has what it needs. Not only is testosterone an essential hormone but it’s also highly valuable in the name of performance. Once we come off our anabolic steroid cycle our natural testosterone will once again be produced but this doesn’t happen overnight; it can take quite a bit of time for the body to recover and normalize. This is where the SERM Nolvadex comes into play, as a solid Nolvadex post cycle plan can greatly aid in stimulating natural testosterone production thereby increasing our natural production much sooner than it would have without. No, as useful as it is a Nolvadex post cycle plan will not return our levels to normal in a mere few weeks but it will send you well on your way and aid in making the transition to off cycle much smoother.

Nolvadex Post Cycle Plan:

As a solid Nolvadex post cycle plan can greatly aid your total recovery process the most obvious question is how much do you need and of course how long do you need to take it? Generally for most men 40mg of Nolvadex per day for a couple of weeks followed by a few weeks at 20mg per day is about all they will need; there are other things you can add to the equation but that should take care of your Nolvadex post cycle needs. A solid plan would look as follows:

Week 1: 40mg every day

Week 2: 40mg every day

Week 3: 20mg every day

Week 4: 20mg every day

While the above will take care of most there are those who will need a little more and unfortunately without your personal information we cannot determine if you will be one of those or not. Some men may indeed need as much as 5-6 weeks of total Nolvadex post cycle therapy. A sample plan might look something like this:

Week 1: 40mg every day

Week 2: 40mg every day

Week 3: 40mg every day

Week 4: 20mg every day

Week 5: 20mg every day

Week 6: 20mg every day

Enhancing your Nolvadex Post Cycle Therapy:

As recovery is so very important and while some anabolic steroid cycles will only require use of a SERM there are additional things you can add. It should be noted you do not have to supplement with Nolvadex post cycle to meet your needs, another quality option is Clomiphene Citrate (Clomid) and this same post cycle enhanced plan will work there as well. To enhance you Nolvadex post cycle therapy the best addition you can make is Human Chorionic Gonadotropin (hCG) and this can really take your post cycle from good to great. Generally the hCG therapy will take place before the Nolvadex therapy begins; approximately 10 days of hCG use at a dosing of 1,000iu per day will suffice. Once the hCG use is complete this is when the Nolvadex post cycle therapy will begin.

The Bottom Line:

Recovery after a cycle is something a lot of people don’t like thinking about simply because it’s not as exciting as the actual steroid cycle. Of course so many complain about losing gains after they come off and again, while other factors come into play things like a Nolvadex post cycle plan can be of a great importance. It should be noted if you supplement with hCG as well you will need to time this as you will with your total post cycle therapy. For example, if your steroid cycle ends with any long ester base steroids you will wait 10 days before you start your hCG therapy; if you are not using hCG you will wait 2-3 weeks before beginning Nolvadex therapy. If your cycle ends with all short ester base steroids you will start your post cycle recovery a day or two after your last steroid injection regardless of using hCG or not.

Nolvadex vs. Clomid for Post Cycle Therapy

While practically similar compounds in structure, few people ever really consider Clomid and Nolvadex to be similar. Its not just a common myth in steroid circles, but even in the medical community. This misconception originates from their completely different uses. Nolvadex is most commonly used for the treatment of breast cancer in women, while clomid is generally considered a fertility aid. In bodybuilding circles, from day one, clomid has generally been used as post-cycle therapy and Nolvadex as an anti-estrogen.

But as I intend to demonstrate this is in essence the same. I believe the myth to have originated because Nolvadex is clearly a more powerful anti-estrogen, and the people selling clomid needed another angle to sell the stuff, so it was mostly used as a post-cycle aid. But few users really kamagra understand how clomid (and also Nolvadex, logically) works to bring back natural testosterone in the body after the conclusion of a cycle of androgenic anabolic steroids. After a cycle is over, the level of androgens in the body drop drastically. The body compensates with an overproduction of estrogen to keep steroid levels up. Estrogen as well inhibits the production of natural testosterone, and in the period between the return of natural testosterone and the end of a cycle, a lot of mass is lost. So its in everybody's best interest to bring back natural test as soon as humanly possible. Clomid and Nolvadex will reduce the post-cycle estrogen, so that a steroid deficiency is constant and the hypothalamus is stimulated to regenerate natural testosterone production in the body. That's basically how the mechanism works, nothing more, nothing less.

Both compounds are structurally alike, classified as triphenylethylenes. Nolvadex is clearly the stronger component of the two as it can achieve better results in decreasing overall estrogen with 20-40 mg a day, than clomid can in doses of 100-150 mg a day. A noteworthy difference. Triphenylethylenes are very mild estrogens that do not exert a lot, if any activity at the estrogen receptor, but are still highly attracted to it. As such they will occupy the receptor and keep it from binding estrogens. This means they do not actively work to reduce estrogen in the body like Proviron, Viratase or Arimidex would (by competing for the aromatase enzyme), but that it blocks the receptor so that any estrogen in the body is basically inert, because it has no receptor to bind to.

This has advantages and disadvantages. The disadvantage is that when use is discontinued, the estrogen level is still the same and new problems will develop much sooner. The advantage is that it works much faster and has results sooner than with an aromatase blocker like Proviron or Arimidex. Therefore, when problems such as gynecomastia occur during a cycle of steroids one will usually start 20 mg/day of Nolva or 100 mg/day of clomid straight away, in conjunction with some Proviron or Arimidex. The proviron or Arimidex will actively reduce estrogen while the clomid or Nolvadex will solve your ongoing problem straight away. This way, when use is discontinued there is no immediate rebound.

So which one should you use? Well personally, I'd have to say Nolvadex. Both as an on-cycle anti-estrogen and a post-cycle therapy. As an anti-estrogen its simply much stronger, demonstrated by the fact that better results are obtained with 20-40 mg than with 100-150 mg of clomid. For post-cycle, this plays a key role as well. It deactivates rebound estrogen much faster and more effective. But most importantly, Nolvadex has a direct influence on bringing back natural testosterone, where as clomid may actually have a slight negative influence. The reason being that Tamoxifen (as in Nolvadex) seems to increase the responsiveness of LH (luteinizing hormone) to GnRH (gonadtropin releasing hormone), whereas clomid seems to decrease the responsiveness a bit1.

Another noteworthy fact about Nolvadex is that it acts more potently as an estrogen in the liver. As you remember, I mentioned that clomiphene and tamoxifen are basically weak estrogens. Well, tamoxifen is apparently still quite potent in the liver. This offers us the positive benefits of this hormone in the liver, while avoiding its negative effects elsewhere in the body. As such Nolvadex can have a very positive impact on negative cholesterol levels2 in the body, and therefore too should be considered a better choice than clomid. It will not solve the problem of bad cholesterol levels during Steroid use, but will help to contain the problem to a larger degree.

Another reason why I promote the use of Nolvadex over Clomid post-cycle (as if being 3-4 times stronger and having more of a direct effect on restoring natural test wasn't enough) is because it's a lot safer. Not just because it improves lipid profiles, but also because it simply doesn't have the intrinsic side-effects that Clomid has. Clomid causes more acne for sure, but that's mainly because you need to use a 3-4 times higher dose. But Clomid seems to also affect the eyesight. Long-term clomid therapy causes irreversible changes in eyesight3 in users. Irreversible. For me that alone is reason enough to prefer Nolvadex.

Lastly, one should be aware that use of these compounds can reduce the gains kamagra made on steroids. Nolvadex more so than clomid, simply because it is stronger. Estrogen is responsible for a number of anabolic factors such as increasing growth hormone output, upgrading the androgen receptor and improving glucose utilization. This is why aromatizing steroids like testosterone are still best suited for maximum muscle gain. When reducing the estrogen levels, we therefore reduce the potential gains being made. For this reason one may opt to try clomid during a cycle instead of Nolvadex. Although I would imagine that the problem that needed solved would be of more concern, in which case Nolva remains the weapon of choice. It's a plain fact that there is a high correlation between gains and side-effects. Either you go for maximum gains and tolerate the side-effects, or you reduce the side-effects, and with it the gains. That's life, nothing is free.

Stacking and Use:

If problems of Gynecomastia or other estrogen related symptoms tend to pop up during a cycle the use of 20-30 mg of Nolvadex or 100 mg of Clomid daily should easily contain the problem, and be used until a few days after the problem subsides. For best results and the least amount of problems upon cessation it is best stacked with Proviron (50 mg) or arimidex (0.5 mg) for this duration as well. Its not advised that these products be ran concomitantly with the steroid for the entire duration of the stack, as this will reduce your gains. Instead cease the usage of anti-estrogens once the problem is contained, and should the problem resurface, simply recommence the use of the products in the same manner as described above.

Once a cycle of steroids is concluded one should always initiate a post-cycle therapy to help bring back natural testosterone as soon as possible. This will help you to retain the mass you gained. How this is done depends highly on the type of steroid used. If only orals were used, therapy should start immediately, even the last day of the stack. If short-acting esters or water-based injectables were used, therapy should commence within 4-7 days after last injection, and if long-acting esters were used then it should commence 1.5 to 2 weeks after the last injection was given. The length of the therapy will vary as well, from 3-5 weeks. The longer acting the product was, the longer therapy should be continued to make sure all suppressive factors are cleared before use of Clomid/Nolvadex is discontinued.

For best results, it is best stacked with HCG (Human Chorionic gonadotrophin), which functions as an LH analog and can help bring testicle size back up. HCG use starts the last week of a cycle, and on from there every 5-6 days (usually 1500-3000 IU) and discontinued 1.5 to weeks prior to the cessation of Nolvadex/clomid. The reason being that HCG itself is also suppressive of natural testosterone and should be out of the body before therapy is over, or it will inhibit natural testicle function. But I can not stress enough that HCG possibly plays a more important role in post-cycle therapy than clomid/Nolvadex. For Clomid and Nolvadex, doses are usually tapered down. Its best to start with 40-50 mg of Nolvadex or 150 mg of Clomid for the first week or the first two weeks, and then finish the program with 20-25 mg of Nolvadex or 100 mg of Clomid for an additional two weeks.

written by BigCat


1 Vermeulen A. Comhaire F. Hormonal effects of an anti-estrogen, tamoxifen, in normal and oligospermic men, Fertil. Ster. 29 (1978) 320-27

2 Bruning PF, Bronfer JMG, Hart AAM, Jong-Bakker M, tamoxifen, serum lipoproteins and cardiovascular risk, Br. J. Cancer 1988 Oct, 58 (4) 497-9

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Post Cycle Therapy

Introduction To Post Cycle Therapy

Post cycle therapy (PCT) is perhaps the most important aspect of anabolic steroid use. The concept of PCT did not exist prior to the late 1980s and early 1990s, as the understanding of the mechanisms by which anabolic steroids affected the body were not completely understood during the 1950s, 1960s, and 1970s. It was during this time period where doctors, scientists, and anabolic steroid users were only beginning to learn about the dynamics of anabolic steroids and how they affect the endocrine system. It was understood since the beginning of anabolic steroid use that the exogenous administration of anabolic steroids resulted in triggering the body’s negative feedback loop of the HPTA (Hypothalamic Pituitary Testicular Axis) and that endogenous Testosterone production would, as a result, become suppressed and/or shut down. Unfortunately during the early periods of anabolic steroid use (between the 1950s and 1990), there was limited access to any compounds or knowledge as to how to effectively mitigate this effect.

Today it is a very different story, where scientific and medical understanding of anabolic steroid use has soared exponentially since the old ‘golden era’ days of bodybuilding and anabolic steroid use in athletics. Countless developments of beneficial compounds for the purpose of hormonal recovery after anabolic steroid use, alongside the increased scientific and medical knowledge, has enabled anabolic steroid use and its associated endocrine disruptions to become a far safer endeavor than it ever once was. With the proper knowledge of how to properly and efficiently recover the body’s HPTA and hormonal systems through post cycle therapy (PCT), individuals can not only emerge from their anabolic steroid cycles while holding on to almost all of their muscle gains, but they can also increase the chances upwards of the 90% or higher range of emerging with a fully functional endocrine system and a healthy HPTA.

Following the use of exogenous anabolic steroids, the majority of users will experience what has been dubbed a "hormonal crash" or "post cycle crash", which is a bodily environment in which key hormones essential to the retention of the newly created muscle mass has been suppressed or shut down. The key hormones in question are LH (Luteinizing Hormone), FSH (Follicle Stimulating Hormone), and subsequently (and most importantly), Testosterone. LH and FSH are known as gonadotropins, which are hormones that signal the gonads (testes) to begin or increase the manufacture and secretion of Testosterone. Alongside low levels of these hormones, the overall balance of total hormones will be essentially thrown off, whereby Testosterone levels will be low, and most of the time (depending on many factors), Estrogen levels will be higher, and levels of Cortisol (a steroid hormone that destroys muscle tissue) will be at normal levels. With Testosterone levels low and Cortisol levels in the normal (or high) range, Cortisol now becomes a threat to the newly created muscle that was created during the recent anabolic steroid cycle (Testosterone properly suppresses and counteracts Cortisol’s catabolic effects on muscle tissue). SHBG (Sex Hormone Binding Globulin) is also a concern here as well, which is a protein that binds to sex hormones (Testosterone) and renders them inactive, essentially ‘handcuffing’ them and preventing them from exerting their effects. SHBG will also normally be elevated during the post-cycle weeks as a result of the supraphysiological levels of androgens from the recent anabolic steroid cycle.

The human body will normally restore this imbalance of hormones and recover its endogenous Testosterone levels on its own over time with no assistance, but studies have demonstrated that without the intervention of Testosterone stimulating agents, this will occur over the course of 1 – 4 months. This is quite evidently enough time for the hormonal imbalance to wreak havoc on the body and result in any individual losing most or all of the newly gained muscle during this time. Therefore, all anabolic steroid users should be concerned with the fastest possible hormonal recovery, assisted and boosted with the use of Testosterone stimulating compounds in the proper manner. Furthermore, the attempt to allow the body to recover on its own will present a very high probability of long-term endocrine damage to the HPTA over time whereby the individual will develop anabolic steroid induced hypogonadism (the inability to manufacture proper levels of Testosterone for the rest of their life). It is therefore paramount that a proper post cycle therapy that includes multiple recovery compounds be utilized so as to not only restore the HPTA function to normal levels as quickly as possible, but to avoid any possible permanent damage, which takes priority over the concern of maintaining the recently gained muscle mass.

What Post Cycle Therapy Protocol To Use? There Are Too Many Of Them!

There exist many different types of PCT protocols that have been developed over the years, and upon first glance, any individual will become extremely confused at how many different opinions exist among the anabolic steroid using community, as well as how many different established PCT protocols there are in existence. This article will present forth the best possible and most efficient post cycle therapy protocol backed by valid scientific data and logical reasoning. This article will also dispel various myths in regards to PCT, and outline which PCT protocols should not be followed due to recent more advanced developments, as well as recent better scientific and medical understandings of how a proper post cycle therapy protocol should work. At this point in time, there still exists very obsolete – and subsequently ineffective – PCT protocols that are still utilized by many anabolic steroid users, and this presents a serious hazard not only for the individual unknowingly using an obsolete post cycle therapy program, but to any individuals that might be observing, learning, and gathering ideas from that individual.

Without the proper understanding of what is specifically occurring within the endocrine system during these crucial weeks, as well as a lack of understanding of which compounds to utilize, what each compound does, and how to properly utilize them, serious problems can result.

The HPTA: How It Works

The HPTA is the Hypothalamic Pituitary Testicular Axis. which is an axis of interconnected endocrine glands in the body that deal with and control Testosterone production.

Outlined above is a diagram of the HPTA. The HPTA regulates how much Testosterone is manufactured and circulating the body at any one given time. Every individual is essentially programmed by their genetics (DNA) as to how much maximum Testosterone they will manufacture, and this is the prime determining factor. There exist other factors that determine how much Testosterone an individual will produce as well, and these include: age, diet, body composition, lifestyle habits, and physical activity. All of these factors play a role in how much Testosterone an individual will generate overall.

The HPTA functions under what is known as the negative feedback loop, whereby the body will reduce its manufacture and secretion of Testosterone if too much Testosterone is detected circulating in the body, and will also adjust as such if insufficient amounts of Testosterone are detected. This detection and adjustment, known as the negative feedback loop, is controlled by the hypothalamus, which is essentially considered the ‘master’ gland for all endocrine and hormonal functions in the body. The negative feedback loop is ultimately the body’s attempt to maintain hormonal homeostasis, which refers to the regulation of a system (in this case, the internal systems of the body) in order to maintain stable and constant favorable conditions. All endocrine glands operate by way of the negative feedback loop in one way or another, and to varying degrees. In the case of post cycle therapy . the concern is primarily with the negative feedback loop of the HPTA.

Within the HPTA, the concern during PCT is the restoration and regulation of the following 5 hormones to homeostasis:

- GnRH (Gonadotropin Releasing Hormone)

- LH (Luteinizing Hormone)

- FSH (Follicle Stimulating Hormone)

- Testosterone

The HPTA begins with the first axis point, the hypothalamus, which will detect a need for the human body to manufacture more Testosterone, and will release varying amounts of GnRH. GnRH is a hormone that signals the next axis point, the pituitary gland, to begin the manufacture and release of two important gonadotropins: LH and FSH. LH and FSH are two hormones that work to signal the third axis point, the testes, to begin production and secretion of Testosterone. This is the final stage of Testosterone production in the HPTA.

There are two primary hormonal factors that serve to inhibit, reduce, suppress, or shut down Testosterone production in the HPTA:

- Excess Testosterone

Although there exist other hormones that serve to inhibit and suppress HPTA function (such as Progestins and Prolactin), these are the two primary conditional hormones that are of concern.  When the hypothalamus detects excess levels of Testosterone and/or Estrogen in the body (either from the use of exogenous androgens on an anabolic steroid cycle or otherwise), the hypothalamus will act to attempt to restore a balance by essentially doing the opposite of what was previously described. The hypothalamus will reduce or stop its production of GnRH, which halts production of LH and FSH, which ultimately reduces or halts production of Testosterone. Until the hypothalamus’ ideal hormonal environment is restored, the production of the various signaling hormones within the HPTA will not begin, and this will often require months of time for the body to do this on its own without the intervention of any Testosterone stimulating agents. The reason as to why the recovery of the HPTA naturally takes such a long time should be very clear due to the described workings of the HPTA.

This very basic understanding of the mechanisms of the HPTA and negative feedback loop described above is essential to understanding how and why a proper PCT program must be developed and utilized following an anabolic steroid cycle .

Determining Factors In Difficulty Recovering the HPTA

With anabolic steroid use, there are several different major determining factors in how much difficulty an individual will experience in recovery of their HPTA and endogenous Testosterone function during PCT. They are the following factors, in no particular order of importance:

1. Individual response

2. Type of anabolic steroid(s) used

3. Length of cycle (degree of testicular desensitization)

1. Individual response: Every single individual will respond in a different manner to any chemical, compound, anabolic steroid, food or drug in existence. While some individuals might experience absolutely no HPTA suppression or shutdown at all, other individuals might experience severe HPTA suppression and shutdown to the extent where they might require far longer periods of time to ensure full recovery than most. This, like anything else, is a spectrum whereby there are the very ‘lucky’ individuals that recover very quickly and easily on one end of the spectrum, and the ‘unlucky’ individuals that have extreme difficulty recovering during post cycle therapy. In between the two extremes is the average. Once again, this is due to the individual’s genetic programming as to how the HPTA will respond and attempt to maintain homeostasis.

2. Type of anabolic steroid(s) used: All anabolic steroids exhibit suppression or shutdown of the HPTA through the mechanisms of the negative feedback loop, and there are no exceptions to this. Various anabolic steroids are known as being mildly suppressive, while others are known as being heavily suppressive. This is all reliant on various different reasons, many of which will not be discussed here. In any case, no matter how mild or severe an anabolic steroid exerts HPTA suppression, all anabolic steroids when utilized for typical cycle lengths of weeks at a time will eventually cause the HPTA to shut down, or at the very least severely suppress its hormonal signal processes.

3. Length of cycle (degree of testicular desensitization): This is perhaps the most important and most influential factor. As the length of anabolic steroid use continues, the majority of the Leydig cells of the testes remain dormant and inactive, and the longer these interstitial cells remain dormant and inactive, the greater the difficulty in essentially getting these cells to respond to the stimulus of LH and FSH once again. It has been discovered in studies that the issue of recovery of the Leydig cells following anabolic steroid use is not due to a lack of LH, but due instead to the desensitization of the Leydig cells to LH[1]. In one study in which exogenous Testosterone was administered to male test subjects for 21 weeks, LH levels were suppressed shortly after beginning administration. However, at the end of the 21 week period, LH levels were observed to rise within 3 weeks once the exogenous Testosterone administration stopped, but Testosterone levels did not rise until many weeks later in most of the test subjects.

The Three Primary Testosterone Stimulating Agents for HPTA Recovery During PCT

Before delving into the three different types of Testosterone stimulating compounds for hormonal recovery during post cycle therapy, it is very important for individuals to understand that the use of any one single compound except for a single select one or two is inadequate for hormonal recovery during PCT. Ideally, all post cycle therapy programs should be a multi-component PCT program that includes several different compounds that work in tandem with one another in order to provide the most effective and fastest possible HPTA recovery following an anabolic steroid cycle.

The three categories of compounds are (in order of importance):

1. SERMs (Selective Estrogen Receptor Modulators)

2. Aromatase Inhibitors

SERMs: Classes of drugs in the SERM category include: Nolvadex (Tamoxifen Citrate), Clomid (Clomiphene Citrate), Raloxifene, and Fareston (Toremifene Citrate). The nature of a SERM is that it exhibits mixed Estrogen agonist and Estrogen antagonist effects on the body. This means that although a SERM might block the effect of Estrogen at the cellular level in certain tissues, it can enhance Estrogenic effects in other areas of the body. These can be positive effects as well as negative effects. Nolvadex, for example, exhibits Estrogenic agonistic effects in the liver, which for all intents and purposes is a positive effect, as its effects here result in a positive change in cholesterol profiles (something desired by many). All SERMs to varying degrees serve to act as an Estrogen antagonist in this area, acting to mitigate Estrogen’s effects on breast tissue, reducing or blocking the side effect of gynecomastia. In terms of the effect of SERMs on endogenous Testosterone stimulation, they serve to act as an Estrogen antagonist at the pituitary gland, triggering the release of LH and FSH as a result. Elevated levels of Estrogen in men can and does suppress the output of endogenous Testosterone via the negative feedback loop, leading to hypogonadism[2]. SERMs for this purpose are an absolutely essential addition to any PCT protocol and are not to be excluded under any circumstance. Regardless of this, however, the sole focus should not be on SERMs.

Aromatase inhibitors: These are compounds such as Aromasin (Exemestane ), Arimidex (Anastrozole ), and Letrozole (Femara). Rather than block the activity of Estrogen at the cellular level in different tissues, aromatase inhibitors (AIs) serve to lower total circulating Estrogen levels in the body by way of inhibiting the aromatase enzyme, which is the enzyme responsible for the conversion of androgens into Estrogen. The conversion of androgens into Estrogen results in excess Estrogen levels, which, as explained earlier in this article, will trigger the negative feedback loop leading to suppression of Testosterone production. By way of lowering total circulating blood plasma Estrogen levels, AIs will engage the negative feedback loop in a positive manner and result in the release of LH and FSH for the manufacture and secretion of more Testosterone. This is essentially due to the hypothalamus realizing that circulating Estrogen levels are too low, and will attempt to increase circulating levels of Testosterone in order for a portion of the Testosterone secreted to be able to become aromatized into Estrogen in order to restore the hormonal balance. The other importance of aromatase inhibitors is the ability to mitigate the Estrogenic effects of HCG, which will be explained shortly. It is important to note, however, that the majority of aromatase inhibitors do not comply very well with SERMs such as Nolvadex, and that very specific choices should be made in regards as to which AI is used during PCT .

HCG: Human Chorionic Gonadotropin is, for the most part, synthetic LH. It is a protein hormone manufactured in high amounts by pregnant females that contains a protein subunit that is 100% identical to LH, and therefore when administered to men, it will mimic the action of LH in target tissues, such as the testes. What results is an increase in Testosterone production via stimulation of the Leydig cells by HCG.  HCG should never be utilized alone, as its nature as a gonadotropin will itself trigger a negative feedback loop whereby once HCG is utilized, the pituitary gland will halt output of LH until HCG use has discontinued. Therefore, HCG must be utilized with a SERM and especially an aromatase inhibitor, as HCG has demonstrated to increase aromatase activity in the testes, resulting in rising Estrogen levels[3] .

Putting Them All Together

The reader may be wondering which compounds to select of the three categories listed, and how to properly use them. The answer lies in understanding the properties of each and, in understanding these properties, how to use them efficiently and appropriately.


The first item to be examined will be HCG. The majority of anabolic steroid users from the 1960s – mid 1980s did not even utilize any compounds for the purpose of hormonal recovery, and the term PCT did not even exist at that time. When the use of HCG became increasingly popular (circa 1980), it was the only compound utilized. Since then, the medical and scientific understanding of such things has increased exponentially and there should be no reason for any informed and properly educated individual to utilize HCG on its own for PCT. When utilized in conjunction with one of the other two categories of compounds (an AI and a SERM), the dynamics change considerably.

It has been mentioned already that much of the difficulty in recovering the HPTA following an anabolic steroid cycle is the result of Leydig cell desensitization. HCG is essentially an analogue of LH, and the testes after a prolonged anabolic steroid cycle would be as equally desensitized to HCG as they are to LH. The human body, however, produces LH amounts on its own that are far too inefficient for proper and rapid Testosterone production. The body’s natural increase of LH and FSH following an anabolic steroid cycle is also not a rapid peak, but a very slow and steady incline, as evidenced by the study referenced earlier in which it was not until 3 weeks when LH levels only began to reach the normal physiological measurements following the cessation of exogenous Testosterone. Therefore, the body’s own natural LH production does not provide a high enough dose for stimulation, nor an immediate stimulation to the testes required for the initial increase in Testosterone needed during the post cycle therapy weeks.

HCG, utilized in a specific manner during the first 1 – 2 weeks of PCT at a dose of 100-1,500IU every 2 days, is what allows the individual to provide the testes with a high dose to provide them with a ‘shock’ effect, and sustain this shock effect on the Leydig cells of the testes for a sustained period of the first 1 – 2 weeks of post cycle therapy. Studies have in fact demonstrated the incredible effectiveness of HCG for this purpose, and it is even suggested clinically that HCG be utilized for the purpose of treating anabolic steroid induced hypogonadism[4]. Following this line of thought, the other two compounds (the SERM and the AI ) are to be utilized as supportive compounds for HCG use in this 1 – 2 week period, and after HCG is discontinued early on in PCT, only the SERM is to be used in order to carry along the hormonal recovery process.

In spite of the good news in regards to the ability for HCG to assist in hormonal recovery, there are still two remaining issues to be addressed:

- The fact that HCG causes increased production of aromatase, leading to increased Estrogen levels.

- Following the discontinuation of HCG, the body is left with very little endogenous LH and FSH production due to the exogenous administration of HCG.

Aromatase Inhibitors: Aromasin (Exemestane) Above All Else

The first of the two remaining issues to be addressed will be the fact that HCG will trigger increases in testicular aromatase expression, and result in Estrogen increases in the body. It should also be noted that it will cause an increase in testicular progesterone levels. Estrogen rising is of course undesirable during PCT, as it has already been explained that Estrogen will trigger suppression of endogenous Testosterone production, and there is no doubt that any individual wishes to encounter Estrogenic side effects during PCT either.

Therefore, the option here is to include an aromatase inhibitor. However, there exists a big problem in regards to the other two of the three major aromatase inhibitors (Arimidex and Letrozole). The issue is the fact that in a PCT program that includes the use of SERMs such as Nolvadex and Clomid, which are known as absolutely essential components to a PCT program, Arimidex and Letrozole have direct negative interactions with Nolvadex. The problem here is that Arimidex (or Letrozole) and Nolvadex both directly counteract one another. One study has demonstrated that when Arimidex is utilized with Nolvadex, Nolvadex will decrease blood plasma concentration of Arimidex (as well as Letrozole, another commonly used aromatase inhibitor)[5]. The conclusion here is that the use of Arimidex or Letrozole with Nolvadex together is a very bad idea and may work counterproductively if used together in a PCT protocol. Aromasin completely circumvents this problem, as it has been demonstrated to have no interactions what so ever with Nolvadex, unlike the other two aforementioned aromatase inhibitors. In one study, Aromasin displayed no such reduced effectiveness or any reduced blood plasma levels when utilized with Nolvadex[6] .

The other benefit of selecting Aromasin over all other AIs is the fact that Aromasin has demonstrated in several studies to impact cholesterol profiles in a negative manner far less than other aromatase inhibitors have, where in one particular study on cancer patients, 24 weeks of Aromasin (Exemestane) administration held no impact on cholesterol profiles[7]. Some other studies have also demonstrated a nil effect on cholesterol profiles from the use of Aromasin[8]. Although there have also been some studies that have demonstrated a negative effect on cholesterol profiles resultant from Aromasin use, it is evident that there is not as a significant or as a negatively impacting effect from Aromasin on cholesterol as other aromatase inhibitors[9] .

Finally, in addition to these benefits from Aromasin, it is very clear that Aromasin holds the ability to increase Testosterone levels in males as demonstrated by studies. For example, one particularly notable study selected 12 healthy young male test subjects, and were administered random Aromasin doses of 25mg and 50mg for a 10 day period, and not only was Estrogen suppressed by a significant amount (38%), but Testosterone levels in the test subjects were observed to have increased by an incredible 60%[10] .

Following these details, Aromasin would be the best possible aromatase inhibitor of choice in order to combat the increased aromatase activity caused by HCG. Therefore, Aromasin would then be utilized at a full 25mg daily dose, and only while HCG is utilized. Once HCG is discontinued, Aromasin too should be halted.

The only following issue to cover now is that of stimulating and maintaining proper endogenous LH release so as to carry recovery along until the body can become self-sufficient once again.

SERMs: Nolvadex and Clomid

The question is often asked among the anabolic steroid using community: Clomid or Nolvadex? Which one for PCT?

First of all, the best possible addition to HCG in a PCT protocol is Nolvadex (Tamoxifen Citrate ), as studies have demonstrated that HCG and Nolvadex utilized together have exhibited a remarkable synergistic effect in terms of stimulating endogenous Testosterone production, and that Nolvadex will actually work to block the desensitization effect on the Leydig cells of the testes caused by high doses of HCG[11]. This is very important, because just as too little LH secretion for extended periods can cause desensitization to gonadotropins, too much gonadotropin stimulation (in the form of HCG or otherwise) will likewise cause a desensitization effect.

Secondly, Nolvadex on a mg for mg basis is far more effective than Clomid in stimulating endogenous Testosterone production, as well as being a more cost-effective choice than Clomid itself. Studies have demonstrated that 150mg of Clomid (Clomiphene Citrate) administered daily raised endogenous Testosterone levels of 10 healthy males by approximately 150%, while incidentally, 20mg of Nolvadex (Tamoxifen Citrate) daily raised endogenous Testosterone levels by the same amount[12]. It is very evident here that Clomid is very effective for this purpose, but Nolvadex seems to be a more cost-effective choice seeing as though it is more effective than Clomid when compared mg for mg. The benefits of Nolvadex over Clomid do not end there – Clomid, although it does exhibit Estrogen antagonist effects at the pituitary gland like Nolvadex does, actually exhibits Estrogen agonist effects there too[13]. What this means is that Clomid will actually work in varying degrees as an Estrogen at the pituitary gland, triggering the negative feedback loop and reducing the output of Testosterone stimulating gonadotropins (LH and FSH). This is a very serious problem during post cycle therapy, which is a period in which individuals are trying to recover their HPTA function rather than halt it even further. Ideally, one would want a SERM that exhibits almost 100% Estrogen antagonistic effects on the pituitary gland, and Nolvadex is the perfect choice for this.

When it comes to the dosing aspect of Nolvadex, The standard dose for PCT and for stimulating the release of GnRH (Gonadotropin Releasing Hormone), LH, FSH, and ultimately Testosterone is that of a simple Nolvadex dose of 20 – 40mg daily. In all studies involving Nolvadex doses used to stimulate endogenous Testosterone production, only 20 – 40mg daily of Nolvadex was utilized, and it has in fact been shown that doubling the dose to 40mg or any higher will not produce any significant difference in endogenous Testosterone secretion. The only reason why many elect to utilize 40mg daily of Nolvadex for the first 1-2 weeks of a PCT program is for the purpose of achieving optimal peak blood plasma levels quicker so as to ensure HPTA recovery quicker.

The Final Layout

The ideal post cycle therapy protocol should then be as follows:

4 – 6 weeks Total PCT time (depending on recovery ability of the individual)

Weeks 1 – 2:

- HCG at 1000iu/E2D

- Aromasin (Exemestane) at 25mg/day

- Nolvadex (Tamoxifen Citrate) at 40mg/day

Weeks 2 – 6:

- Nolvadex (Tamoxifen Citrate) at 20mg/day

Additional Optional Components (Vitamins/Supplements/Compounds) to Aid During PCT

Vitamin D (Cholecalciferol): There is plenty of established solid evidence through studies that mega dosing Vitamin D (Cholecalciferol) exhibits a significant effect on increasing Testosterone levels in men and also has a significant ability to suppress SHBG levels in the body. One of the best PCT additions is indeed Vitamin D. There is an overabundance of clinical studies in existence demonstrating that low Vitamin D levels corresponds with a low level of endogenous Testosterone production (particularly in the winter months for obvious reasons). In one study conducted in Austria where about 200 subjects were involved with one group administered 3332iu daily of vitamin D, and a placebo group, results had shown that men with sufficient Vitamin D levels had significantly higher levels of Testosterone and significantly lower levels of SHBG when compared to the D-deficient subjects[14]. Androgen levels and Vitamin D levels are associated in men and reveal a concordant seasonal variation[15]. In various other studies, similar findings were reported where subjects who were administered higher amounts of Vitamin D over time demonstrated vast increases in total Testosterone levels, and decreases in SHBG. Anecdotal evidence of people who have been supplementing with vitamin D and getting regular blood work at their doctors are observing large increases in their total and free testosterone levels approximately 1 – 2 months after supplementing with Vitamin D.


The normal route for post cycle is to take an anti-estrogen like ATD in hopes that you will get faster recovery. Also people may take a SERM like Nolvadex to lower the estrogen levels in the pituitary.  The major problem is that all this accomplishes is to send more signal.  Remember the testicles are like an engine and the pituitary is like a gas pedal. By lowering estrogen, you are just sending out more signal, but this only works up to a point.

After a cycle, your estrogen and DHT levels are already at a low point due to the natural shut down.  So, ther is no need to “lower” them with a SERM or Aromatase Inhibitor.  The pituitary already sees that the levels are low and is sending out tons of signal.

The best approach is to increase the performance of the engine in addition to stepping on the pedal. You do that with supplements that increase the enzyme activity in the testicles. The best three supplements for this are Forskolin, Ginger Root and D-Aspartic Acid.  These three supplements will surely boost your testosterone production after a cycle. Forskolin increases cAMP levels, causing your testicles to be more efficient at making testosterone. Ginger and D-Aspartic Acid increase StAR synthesis, which is another enzyme in the testicles responsible for making androgens.  By stacking these with an anti-estrogen you get the benefit of increased signal along with a boost in testicular efficiency.

The combination is the most effective post cycle therapy protocol.  Additionally, you may want to take additional fish oil to help lower the potential blood pressure and cholesterol effects of a steroid cycle, but short term steroid or prohormone use shouldn’t have much of an effect long term as long as you have naturally low cholesterol and blood pressure levels.

Ready To Try A Prohormone Cycle?

This 30-Day Basic Cutting stack is available only at LeanBulk and contains all you need for a mass-building cycle plus post-cycle therapy. Also check out our Basic Mass Stack .

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All the informations needed to do an OTC PCT.

What is an OTC PCT – Over The Counter Post Cycle Therapy ?

An OTC PCT is a little bit different than a normal Post cycle therapy. Instead of using SERMs, we use over the counter post cycle therapy supplements. These supplements can be bought pretty much in any bodybuilding stores. This is why we call it “over the counter”.

What is the best Post cycle therapy, a PCT with SERMs or an OTC PCT ?

Without any doubt, the best post cycle therapy is with SERMs but with some prohormones, an OTC PCT can be enough. The SERMs are the best products to use for a post cycle therapy because the Otc Pct supplements are way less potent. However, the Otc Pct supplements are still enough potent so we can use them for many prohormone cycles.

What are the prohormones I can and can’t take when doing an OTC PCT ?

Prohormones not eligible for an OTC PCT :

Superdrol, Pheraplex, M-1,4ADD. Max LMG, Dymethazine, M-Sten, Cynostane, Methyl-1-Alpha

Prohormones eligible for an OTC PCT :

Propadrol, 11-OXO, Bold, Halodrol, Promagnon, Protodrol, Tren, Epistane-Havoc, Furazadrol, 1-Andro, D-Plex, Prostanozol, Stanodrol

What are the bodybuilding supplements needed to do a proper OTC PCT ?

To do a proper OTC PCT, you will need to take multiple supplements. Why. To obtain a similar effect to a SERM, you have to combine the OTC PCT/Post cycle therapy supplements. Remind you that the Post Cycle Therapy supplements are here to help boost natural production of testosterone and to control the level of estrogen after a cycle of prohormone.

A testosterone booster :

You need a good testosterone that will boost your natural production of testosterone + control your estrogen level. A good testosterone booster is generally composed with Divinal and Indole-3-Carbinol (I3C).

* You can read our reviews and reviews from users of Testosterone Boosters by clicking here. 

Steroid Ancillaries | Post Cycle Therapy | Kalpa Pharmaceuticals Anti - Etrogens | Buy Steroid Post Cycle Drugs


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